Walter Reed Woes Greater than Bldg 18

Didn't post this within other article for fear y'all would miss;
http://prorev.com/2007/03/walter-reed-damage-throughout-whole.htm

Be sure to view the video link at bottom of article.

Sailor's family finds how dangerous mold can be in...

Nevada.

*VIKTORIA PEARSON, vpearson@lahontanvalleynews.com
April 8, 2007

How much can one mother take is a question Michelle Bodenhorn of Fallon continues to ask herself each day while she sits in her 2-year-old daughter Elizabeth's hospital room at Renown Regional Medical Center in Reno.

Bodenhorn was told in February that she needed to leave her home and all the family's possessions behind because of a mold problem caused by a water leak. *

http://www.lahontanvalleynews.com/article/20070408/News/104080040

Stages of Mycotoxicosis: For Inhalation of Mycotoxin

Note the date on this paper.

Stages of Mycotoxicosis: For Inhalation of Mycotoxin
By Dr. William Croft

The three Stages (1-3) ranging from lower to higher
severity of poisoning were modified according to exposure
via the air as opposed to ingestion already established
(Forgacs et al., 1962; Joffe, 1971). A separate Stage of
convalescence occurs when a patient is completely removed
from the contaminated premises and the source of mycotoxin
or mold spores.

Stage 1: The primary changes are in the brain, respiratory
and immune systems, mucus membranes and gastrointestinal
tract. Signs and symptoms may include burning sensation in
the mouth, tongue, throat, palate, esophagus, and stomach,
which is a result of the action of the toxin on the mucous
membranes and skin in the exposed areas. Moist areas of
the body armpits, under breasts, belt line and groin are
more sensitive or first affected. Patients may report
burning within the eyes, ears and nose. Patients also
reported that their tongues felt swollen and stiff. Mucosa
of the oral cavity may be hyperemic. Mild gingivitis,
stomatitis, glositis, and esophagitis developed.
Inflammation, in addition to gastric and (small and large)
intestinal mucosal, resulted in vomiting, diarrhea and
abdominal pain. Excessive salivation, headache, dizziness,
weakness, fatigue and tachycardia were also present.

There may be fever and sweating. The respiratory system
develops burning sensations and congestion. Severe
exposure to mycotoxin within the lungs may lead to
congestion, edema and failure, due to caustic action. Body
temperature remains normal and controllable by the
patient. The poisoning appears and disappears relatively
quickly in this Stage with the exception of, lungs and
central nervous system. Initially (Stage 1), the patient�s
symptoms are very uncomfortable or painful. As the
poisoning continues and the patient progress toward Stage
2, he or she becomes accustomed to the presence of the
mycotoxin and a quiescent period follows due to lack of
nerve sensation. Depending on exposure levels, the first
Stage may last from 3 - 9 days. In scoring the 50 signs
and symptoms listed in Tables-1 and 2, an average score
range of 20-45 represents Stage 1.

Stage 2 : This Stage is often called the latent Stage or
incubation period because the patient feels apprehensive,
but is capable of normal activity in the beginning of this
Stage. Every organ of the body is affected by degeneration
and necrosis with continued exposure. The primary target
organs for an individual become evident over time, due to
biological variation. These are disturbances in the
central and autonomic nervous systems resulting in
headaches, mental depression, loss of short-term memory,
loss of problem-solving ability, various neuropsychiatric
manifestations, meningism, severe malaise and fatigue,
narcolepsy, loss of temperature control, hyperesthesia or
numbness of body areas, and cerebellar dysfunction
including hypotonia, attitude and gait, dysmetria,
asthenia, vertigo, disturbances of speech, and loss of
balance (Best, 1961). Spinal cord degeneration may also be
observed in gait and reflex abnormalities, such as the
ability to drive vehicles, ride bicycles or pass sobriety
tests (inability to tolerate ethyl alcohol). Attention
deficient disorder may be observed in children. Various
systems may include: Eyes: visual disturbances, floating
objects, light sensitive, lack of tears, burning and
itching. Ears: burning, itching, and loss of hearing.
Immune and hematopoietic: progressive loss of white and
red cells including a decrease of platelets and
hemoglobin, and high susceptibility to bacterial, mycotic
and viral infections, debilitating chemical and allergies.
Gastrointestinal: metallic taste in mouth, tooth loss, gum
problems, stomatitis, sores in gums and throat, nausea,
vomiting, diarrhea or constipation, excessive flatulence,
abdominal distention, hepatitis, pancreatitis, and
diabetes mellitus. Respiratory : burning and bleeding from
nasal membranes, respiratory difficulty, asthma, extreme
susceptibility to cold, flu and pneumonia. Skin: thinning
of hair on head, burning on face, rashes, irritation, and
edema. Renal: proteinuria, possible hematuria.
Reproductive: irregular ovarian cycles, increased
menstrual flow, fibroid growths in uterus, cystic
development in mammary glands, and tumors of mammary and
prostate glands. Musculoskeletal : somatitis, muscle
weakness, spasms, cramps, joint pain, enlargement of
joints in hand, and clubbing of fingers. Cardiovascular:
chest pain, palpitations, ruptures of atrial walls,
myocardial infection and aneurysm of arteries.

The skin and mucous membranes may be icteric, pupils
dilated, the pulse soft and labile, and blood pressure may
decrease or increase. The body temperature does not exceed
38 degree C and the patient may be afebrile, or chilled.
Visible hemorrhagic spots may appear on the skin. Thoughts
of suicide may be prominent in the person�s mind at this
time or anytime in Stage 2. Human bonding is very
important for survival.

Degeneration and hemorrhages of the vessels marks the
transition from the second to the third Stage of the
disease and may not be consistently observed. The
degeneration of the vital organs including serious
respiratory insufficiency or asthma and CNS degeneration
will take the patient into Stage three along with
development of necrotic angina. If exposure continues,
depending on exposure levels, Stage 2 may continue from
weeks to months or even years until the symptoms of the
third Stage develop. Evaluating the 50 signs and symptoms
(Table-1 and 2) by assigning a score (0-least intense to 5-
most intense or severe) to each symptom, we have
determined that an average score range of 45-180
represents Stage 2.

Stage 3: Severe degeneration of the vital organs. The
transition from the second to the third Stage is sudden.
In this Stage, the patient�s resistance is already low,
and violent severe symptoms are present, especially under
the influence of stress, or associated with physical
exertion and fatigue. The first visible sign of this Stage
may be lung, brain or heart failure (heart attack), with
or without the appearance of petechial hemorrhage on the
skin of the trunk, the axillary and inguinal areas, the
lateral surfaces of the arms and thighs, the face and
head, and in serious Cases, the chest. The petechial
hemorrhages vary from a few millimeters to a few
centimeters in diameter. There is increased capillary
fragility and any slight trauma may cause the hemorrhages
to increase in size.

Aneurysms of the brain or aorta may be observed by
angiography. Hemorrhages may also be found on the mucous
membranes of the mouth and tongue, and on the soft palate
and tonsils. There may be severe interstitial thickening
or scarring of the lungs, or respiratory failure. Nasal,
gastric and intestinal hemorrhages and hemorrhagic
diathesis may occur. Necrotic angina begins in the form of
catarrhal symptoms and necrotic changes soon appear in the
mouth, throat, and esophagus with difficulty and pain on
swallowing. Severe degeneration of the skin on the face,
eyelids, and loss of lashes is also often present.

Necrotic lesions may extend to the uvula, gums, buccal
mucosa, larynx, vocal cords, lungs, stomach, and
intestines and other internal organs such as the liver and
kidneys and are usually contaminated with a variety of
avirulent bacteria. Bacteria infection causes an
unpleasant odor from the mouth due to the enzymatic
activity of bacteria on proteins. Areas of necrosis may
also appear on the lips and on the skin of the fingers,
nose, jaws, and eyes. Regional lymph nodes are frequently
enlarged. Esophageal lesions may occur and involvement of
the epiglottis may cause laryngeal edema and aphonia (loss
of voice). Death may occur by strangulation.

Patients may suffer an acute parenchymatous hepatitis
accompanied by jaundice. Bronchopneumonia, pulmonary
hemorrhages, and lung abscesses are frequent
complications. Tumors may develop of various organs,
including skin, urinary bladder, brain, mammary gland,
bone, immune, liver, prostate, possibly resulting in
death. The most common cause of death is brain failure due
to both direct effects of the mycotoxin on the central
nervous system and indirect effects due to respiratory
failure or lack of oxygen to the brain caused by the
severe caustic inflammation (fibrinous exudation) reaction
with the lung tissue, rendering it non-functional. Again,
using the scoring system represented in Tables-1 and 2, an
average score of greater or equal 180 represents Stage 3.

Stage of Convalescence: The course and duration of this
Stage 3 depends on the intensity of the poisoning and
complete removal of the patient from the premises or
source of mycotoxin. Therefore, the duration of the
recovery period is variable. There is considerable
cellular necrosis and scarring to all major organs of the
body in which cells will not regenerate, including the
brain, spinal cord, eyes, lung, heart, liver, pancreas,
kidney, adrenal, and blood vessels. If the disease is
diagnosed during the first Stage, hospitalization is
usually unnecessary, but allergies and asthma should be
monitored closely. If the disease is diagnosed during the
second Stage and even at the transition from the second to
third Stages, early hospitalization may preserve the
patient�s life. If however, the disease is only detected
during the third Stage, death cannot be prevented in most
Cases.