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Mold Info

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  • Mold Info

    I believe that one of the reasons some of the illnesses that the Viet Vets, in country, have been so difficult to get a handle on is the synergistic impact of the exposure to the rain agents and exotic molds. The fungii, already opportunistic pathogens, were given a big "come on in" to the body stripped of
    its normal immune defenses due to exposure to the chemical agents.

    This site has links to simply listed, well-known symptoms of mold exposure;
    http://mycotoxicosis.com/.

    Fungii are little understood due to their immense numbers of species and variations within those species. At any given time or stage of their life cycle, these pathogens produce different toxins as do each specific specie produce numerous toxins. Very complicated, but the effects are well known and documented.

    Denver, CO;

    VA Hospital turning away some patients due to aspergillus.
    http://www.9news.com/news/article.aspx?storyid=65931

    Check out the '04 report on the Denver facility. Remember, the hospital knew there were problems with aspergillosis in '01.

    http://www.va.gov/oig/publications/reports-list.asp


    Amazing that the Senate and House Veterans Affairs Committee members and their staffs missed those OIG reports.

    http://veterans.senate.gov/
    http://veterans.house.gov/index.htm

    More Veterans' Hospitals in the news;

    http://www.consortiumnews.com/2007/030807b.html

    http://www.dailymail.com/story/News/...vets+a+tragedy

    Here is one I missed from Amarillo regarding the VA Hospital there;
    http://www.kvii.com/news/news_story.aspx?id=28702

    And another about all VA hospitals and Kiley;
    http://www.mysanantonio.com/opinion/...d.297e5ef.html

    Must read; http://www.boston.com/news/education...est_practices/

    http://www.kaisernetwork.org/daily_r...fm?DR_ID=43509

    http://www.capitalnews9.com/content/...7050&SecID=285
    Last edited by widow1; 03-12-2007, 17:17. Reason: All related to same subject from same contributer.
    Deborah

  • #2
    Walter Reed Woes Greater than Bldg 18

    Didn't post this within other article for fear y'all would miss;
    http://prorev.com/2007/03/walter-ree...hout-whole.htm

    Be sure to view the video link at bottom of article.
    Last edited by widow1; 03-21-2007, 18:50. Reason: missing link (I am ;)
    Deborah

    Comment


    • #3
      Sailor's family finds how dangerous mold can be in...

      Nevada.

      ****VIKTORIA PEARSON, vpearson@lahontanvalleynews.com
      April 8, 2007

      How much can one mother take is a question Michelle Bodenhorn of Fallon continues to ask herself each day while she sits in her 2-year-old daughter Elizabeth's hospital room at Renown Regional Medical Center in Reno.

      Bodenhorn was told in February that she needed to leave her home and all the family's possessions behind because of a mold problem caused by a water leak. ****

      http://www.lahontanvalleynews.com/ar...News/104080040
      Deborah

      Comment


      • #4
        Stages of Mycotoxicosis: For Inhalation of Mycotoxin

        Note the date on this paper.

        Stages of Mycotoxicosis: For Inhalation of Mycotoxin
        By Dr. William Croft

        The three Stages (1-3) ranging from lower to higher
        severity of poisoning were modified according to exposure
        via the air as opposed to ingestion already established
        (Forgacs et al., 1962; Joffe, 1971). A separate Stage of
        convalescence occurs when a patient is completely removed
        from the contaminated premises and the source of mycotoxin
        or mold spores.

        Stage 1: The primary changes are in the brain, respiratory
        and immune systems, mucus membranes and gastrointestinal
        tract. Signs and symptoms may include burning sensation in
        the mouth, tongue, throat, palate, esophagus, and stomach,
        which is a result of the action of the toxin on the mucous
        membranes and skin in the exposed areas. Moist areas of
        the body armpits, under breasts, belt line and groin are
        more sensitive or first affected. Patients may report
        burning within the eyes, ears and nose. Patients also
        reported that their tongues felt swollen and stiff. Mucosa
        of the oral cavity may be hyperemic. Mild gingivitis,
        stomatitis, glositis, and esophagitis developed.
        Inflammation, in addition to gastric and (small and large)
        intestinal mucosal, resulted in vomiting, diarrhea and
        abdominal pain. Excessive salivation, headache, dizziness,
        weakness, fatigue and tachycardia were also present.

        There may be fever and sweating. The respiratory system
        develops burning sensations and congestion. Severe
        exposure to mycotoxin within the lungs may lead to
        congestion, edema and failure, due to caustic action. Body
        temperature remains normal and controllable by the
        patient. The poisoning appears and disappears relatively
        quickly in this Stage with the exception of, lungs and
        central nervous system. Initially (Stage 1), the patient�s
        symptoms are very uncomfortable or painful. As the
        poisoning continues and the patient progress toward Stage
        2, he or she becomes accustomed to the presence of the
        mycotoxin and a quiescent period follows due to lack of
        nerve sensation. Depending on exposure levels, the first
        Stage may last from 3 - 9 days. In scoring the 50 signs
        and symptoms listed in Tables-1 and 2, an average score
        range of 20-45 represents Stage 1.

        Stage 2 : This Stage is often called the latent Stage or
        incubation period because the patient feels apprehensive,
        but is capable of normal activity in the beginning of this
        Stage. Every organ of the body is affected by degeneration
        and necrosis with continued exposure. The primary target
        organs for an individual become evident over time, due to
        biological variation. These are disturbances in the
        central and autonomic nervous systems resulting in
        headaches, mental depression, loss of short-term memory,
        loss of problem-solving ability, various neuropsychiatric
        manifestations, meningism, severe malaise and fatigue,
        narcolepsy, loss of temperature control, hyperesthesia or
        numbness of body areas, and cerebellar dysfunction
        including hypotonia, attitude and gait, dysmetria,
        asthenia, vertigo, disturbances of speech, and loss of
        balance (Best, 1961). Spinal cord degeneration may also be
        observed in gait and reflex abnormalities, such as the
        ability to drive vehicles, ride bicycles or pass sobriety
        tests (inability to tolerate ethyl alcohol). Attention
        deficient disorder may be observed in children. Various
        systems may include: Eyes: visual disturbances, floating
        objects, light sensitive, lack of tears, burning and
        itching. Ears: burning, itching, and loss of hearing.
        Immune and hematopoietic: progressive loss of white and
        red cells including a decrease of platelets and
        hemoglobin, and high susceptibility to bacterial, mycotic
        and viral infections, debilitating chemical and allergies.
        Gastrointestinal: metallic taste in mouth, tooth loss, gum
        problems, stomatitis, sores in gums and throat, nausea,
        vomiting, diarrhea or constipation, excessive flatulence,
        abdominal distention, hepatitis, pancreatitis, and
        diabetes mellitus. Respiratory : burning and bleeding from
        nasal membranes, respiratory difficulty, asthma, extreme
        susceptibility to cold, flu and pneumonia. Skin: thinning
        of hair on head, burning on face, rashes, irritation, and
        edema. Renal: proteinuria, possible hematuria.
        Reproductive: irregular ovarian cycles, increased
        menstrual flow, fibroid growths in uterus, cystic
        development in mammary glands, and tumors of mammary and
        prostate glands. Musculoskeletal : somatitis, muscle
        weakness, spasms, cramps, joint pain, enlargement of
        joints in hand, and clubbing of fingers. Cardiovascular:
        chest pain, palpitations, ruptures of atrial walls,
        myocardial infection and aneurysm of arteries.

        The skin and mucous membranes may be icteric, pupils
        dilated, the pulse soft and labile, and blood pressure may
        decrease or increase. The body temperature does not exceed
        38 degree C and the patient may be afebrile, or chilled.
        Visible hemorrhagic spots may appear on the skin. Thoughts
        of suicide may be prominent in the person�s mind at this
        time or anytime in Stage 2. Human bonding is very
        important for survival.

        Degeneration and hemorrhages of the vessels marks the
        transition from the second to the third Stage of the
        disease and may not be consistently observed. The
        degeneration of the vital organs including serious
        respiratory insufficiency or asthma and CNS degeneration
        will take the patient into Stage three along with
        development of necrotic angina. If exposure continues,
        depending on exposure levels, Stage 2 may continue from
        weeks to months or even years until the symptoms of the
        third Stage develop. Evaluating the 50 signs and symptoms
        (Table-1 and 2) by assigning a score (0-least intense to 5-
        most intense or severe) to each symptom, we have
        determined that an average score range of 45-180
        represents Stage 2.

        Stage 3: Severe degeneration of the vital organs. The
        transition from the second to the third Stage is sudden.
        In this Stage, the patient�s resistance is already low,
        and violent severe symptoms are present, especially under
        the influence of stress, or associated with physical
        exertion and fatigue. The first visible sign of this Stage
        may be lung, brain or heart failure (heart attack), with
        or without the appearance of petechial hemorrhage on the
        skin of the trunk, the axillary and inguinal areas, the
        lateral surfaces of the arms and thighs, the face and
        head, and in serious Cases, the chest. The petechial
        hemorrhages vary from a few millimeters to a few
        centimeters in diameter. There is increased capillary
        fragility and any slight trauma may cause the hemorrhages
        to increase in size.

        Aneurysms of the brain or aorta may be observed by
        angiography. Hemorrhages may also be found on the mucous
        membranes of the mouth and tongue, and on the soft palate
        and tonsils. There may be severe interstitial thickening
        or scarring of the lungs, or respiratory failure. Nasal,
        gastric and intestinal hemorrhages and hemorrhagic
        diathesis may occur. Necrotic angina begins in the form of
        catarrhal symptoms and necrotic changes soon appear in the
        mouth, throat, and esophagus with difficulty and pain on
        swallowing. Severe degeneration of the skin on the face,
        eyelids, and loss of lashes is also often present.

        Necrotic lesions may extend to the uvula, gums, buccal
        mucosa, larynx, vocal cords, lungs, stomach, and
        intestines and other internal organs such as the liver and
        kidneys and are usually contaminated with a variety of
        avirulent bacteria. Bacteria infection causes an
        unpleasant odor from the mouth due to the enzymatic
        activity of bacteria on proteins. Areas of necrosis may
        also appear on the lips and on the skin of the fingers,
        nose, jaws, and eyes. Regional lymph nodes are frequently
        enlarged. Esophageal lesions may occur and involvement of
        the epiglottis may cause laryngeal edema and aphonia (loss
        of voice). Death may occur by strangulation.

        Patients may suffer an acute parenchymatous hepatitis
        accompanied by jaundice. Bronchopneumonia, pulmonary
        hemorrhages, and lung abscesses are frequent
        complications. Tumors may develop of various organs,
        including skin, urinary bladder, brain, mammary gland,
        bone, immune, liver, prostate, possibly resulting in
        death. The most common cause of death is brain failure due
        to both direct effects of the mycotoxin on the central
        nervous system and indirect effects due to respiratory
        failure or lack of oxygen to the brain caused by the
        severe caustic inflammation (fibrinous exudation) reaction
        with the lung tissue, rendering it non-functional. Again,
        using the scoring system represented in Tables-1 and 2, an
        average score of greater or equal 180 represents Stage 3.

        Stage of Convalescence: The course and duration of this
        Stage 3 depends on the intensity of the poisoning and
        complete removal of the patient from the premises or
        source of mycotoxin. Therefore, the duration of the
        recovery period is variable. There is considerable
        cellular necrosis and scarring to all major organs of the
        body in which cells will not regenerate, including the
        brain, spinal cord, eyes, lung, heart, liver, pancreas,
        kidney, adrenal, and blood vessels. If the disease is
        diagnosed during the first Stage, hospitalization is
        usually unnecessary, but allergies and asthma should be
        monitored closely. If the disease is diagnosed during the
        second Stage and even at the transition from the second to
        third Stages, early hospitalization may preserve the
        patient�s life. If however, the disease is only detected
        during the third Stage, death cannot be prevented in most
        Cases.
        Deborah

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