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Mold Info

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  • widow1
    replied
    Stages of Mycotoxicosis: For Inhalation of Mycotoxin

    Note the date on this paper.

    Stages of Mycotoxicosis: For Inhalation of Mycotoxin
    By Dr. William Croft

    The three Stages (1-3) ranging from lower to higher
    severity of poisoning were modified according to exposure
    via the air as opposed to ingestion already established
    (Forgacs et al., 1962; Joffe, 1971). A separate Stage of
    convalescence occurs when a patient is completely removed
    from the contaminated premises and the source of mycotoxin
    or mold spores.

    Stage 1: The primary changes are in the brain, respiratory
    and immune systems, mucus membranes and gastrointestinal
    tract. Signs and symptoms may include burning sensation in
    the mouth, tongue, throat, palate, esophagus, and stomach,
    which is a result of the action of the toxin on the mucous
    membranes and skin in the exposed areas. Moist areas of
    the body armpits, under breasts, belt line and groin are
    more sensitive or first affected. Patients may report
    burning within the eyes, ears and nose. Patients also
    reported that their tongues felt swollen and stiff. Mucosa
    of the oral cavity may be hyperemic. Mild gingivitis,
    stomatitis, glositis, and esophagitis developed.
    Inflammation, in addition to gastric and (small and large)
    intestinal mucosal, resulted in vomiting, diarrhea and
    abdominal pain. Excessive salivation, headache, dizziness,
    weakness, fatigue and tachycardia were also present.

    There may be fever and sweating. The respiratory system
    develops burning sensations and congestion. Severe
    exposure to mycotoxin within the lungs may lead to
    congestion, edema and failure, due to caustic action. Body
    temperature remains normal and controllable by the
    patient. The poisoning appears and disappears relatively
    quickly in this Stage with the exception of, lungs and
    central nervous system. Initially (Stage 1), the patient�s
    symptoms are very uncomfortable or painful. As the
    poisoning continues and the patient progress toward Stage
    2, he or she becomes accustomed to the presence of the
    mycotoxin and a quiescent period follows due to lack of
    nerve sensation. Depending on exposure levels, the first
    Stage may last from 3 - 9 days. In scoring the 50 signs
    and symptoms listed in Tables-1 and 2, an average score
    range of 20-45 represents Stage 1.

    Stage 2 : This Stage is often called the latent Stage or
    incubation period because the patient feels apprehensive,
    but is capable of normal activity in the beginning of this
    Stage. Every organ of the body is affected by degeneration
    and necrosis with continued exposure. The primary target
    organs for an individual become evident over time, due to
    biological variation. These are disturbances in the
    central and autonomic nervous systems resulting in
    headaches, mental depression, loss of short-term memory,
    loss of problem-solving ability, various neuropsychiatric
    manifestations, meningism, severe malaise and fatigue,
    narcolepsy, loss of temperature control, hyperesthesia or
    numbness of body areas, and cerebellar dysfunction
    including hypotonia, attitude and gait, dysmetria,
    asthenia, vertigo, disturbances of speech, and loss of
    balance (Best, 1961). Spinal cord degeneration may also be
    observed in gait and reflex abnormalities, such as the
    ability to drive vehicles, ride bicycles or pass sobriety
    tests (inability to tolerate ethyl alcohol). Attention
    deficient disorder may be observed in children. Various
    systems may include: Eyes: visual disturbances, floating
    objects, light sensitive, lack of tears, burning and
    itching. Ears: burning, itching, and loss of hearing.
    Immune and hematopoietic: progressive loss of white and
    red cells including a decrease of platelets and
    hemoglobin, and high susceptibility to bacterial, mycotic
    and viral infections, debilitating chemical and allergies.
    Gastrointestinal: metallic taste in mouth, tooth loss, gum
    problems, stomatitis, sores in gums and throat, nausea,
    vomiting, diarrhea or constipation, excessive flatulence,
    abdominal distention, hepatitis, pancreatitis, and
    diabetes mellitus. Respiratory : burning and bleeding from
    nasal membranes, respiratory difficulty, asthma, extreme
    susceptibility to cold, flu and pneumonia. Skin: thinning
    of hair on head, burning on face, rashes, irritation, and
    edema. Renal: proteinuria, possible hematuria.
    Reproductive: irregular ovarian cycles, increased
    menstrual flow, fibroid growths in uterus, cystic
    development in mammary glands, and tumors of mammary and
    prostate glands. Musculoskeletal : somatitis, muscle
    weakness, spasms, cramps, joint pain, enlargement of
    joints in hand, and clubbing of fingers. Cardiovascular:
    chest pain, palpitations, ruptures of atrial walls,
    myocardial infection and aneurysm of arteries.

    The skin and mucous membranes may be icteric, pupils
    dilated, the pulse soft and labile, and blood pressure may
    decrease or increase. The body temperature does not exceed
    38 degree C and the patient may be afebrile, or chilled.
    Visible hemorrhagic spots may appear on the skin. Thoughts
    of suicide may be prominent in the person�s mind at this
    time or anytime in Stage 2. Human bonding is very
    important for survival.

    Degeneration and hemorrhages of the vessels marks the
    transition from the second to the third Stage of the
    disease and may not be consistently observed. The
    degeneration of the vital organs including serious
    respiratory insufficiency or asthma and CNS degeneration
    will take the patient into Stage three along with
    development of necrotic angina. If exposure continues,
    depending on exposure levels, Stage 2 may continue from
    weeks to months or even years until the symptoms of the
    third Stage develop. Evaluating the 50 signs and symptoms
    (Table-1 and 2) by assigning a score (0-least intense to 5-
    most intense or severe) to each symptom, we have
    determined that an average score range of 45-180
    represents Stage 2.

    Stage 3: Severe degeneration of the vital organs. The
    transition from the second to the third Stage is sudden.
    In this Stage, the patient�s resistance is already low,
    and violent severe symptoms are present, especially under
    the influence of stress, or associated with physical
    exertion and fatigue. The first visible sign of this Stage
    may be lung, brain or heart failure (heart attack), with
    or without the appearance of petechial hemorrhage on the
    skin of the trunk, the axillary and inguinal areas, the
    lateral surfaces of the arms and thighs, the face and
    head, and in serious Cases, the chest. The petechial
    hemorrhages vary from a few millimeters to a few
    centimeters in diameter. There is increased capillary
    fragility and any slight trauma may cause the hemorrhages
    to increase in size.

    Aneurysms of the brain or aorta may be observed by
    angiography. Hemorrhages may also be found on the mucous
    membranes of the mouth and tongue, and on the soft palate
    and tonsils. There may be severe interstitial thickening
    or scarring of the lungs, or respiratory failure. Nasal,
    gastric and intestinal hemorrhages and hemorrhagic
    diathesis may occur. Necrotic angina begins in the form of
    catarrhal symptoms and necrotic changes soon appear in the
    mouth, throat, and esophagus with difficulty and pain on
    swallowing. Severe degeneration of the skin on the face,
    eyelids, and loss of lashes is also often present.

    Necrotic lesions may extend to the uvula, gums, buccal
    mucosa, larynx, vocal cords, lungs, stomach, and
    intestines and other internal organs such as the liver and
    kidneys and are usually contaminated with a variety of
    avirulent bacteria. Bacteria infection causes an
    unpleasant odor from the mouth due to the enzymatic
    activity of bacteria on proteins. Areas of necrosis may
    also appear on the lips and on the skin of the fingers,
    nose, jaws, and eyes. Regional lymph nodes are frequently
    enlarged. Esophageal lesions may occur and involvement of
    the epiglottis may cause laryngeal edema and aphonia (loss
    of voice). Death may occur by strangulation.

    Patients may suffer an acute parenchymatous hepatitis
    accompanied by jaundice. Bronchopneumonia, pulmonary
    hemorrhages, and lung abscesses are frequent
    complications. Tumors may develop of various organs,
    including skin, urinary bladder, brain, mammary gland,
    bone, immune, liver, prostate, possibly resulting in
    death. The most common cause of death is brain failure due
    to both direct effects of the mycotoxin on the central
    nervous system and indirect effects due to respiratory
    failure or lack of oxygen to the brain caused by the
    severe caustic inflammation (fibrinous exudation) reaction
    with the lung tissue, rendering it non-functional. Again,
    using the scoring system represented in Tables-1 and 2, an
    average score of greater or equal 180 represents Stage 3.

    Stage of Convalescence: The course and duration of this
    Stage 3 depends on the intensity of the poisoning and
    complete removal of the patient from the premises or
    source of mycotoxin. Therefore, the duration of the
    recovery period is variable. There is considerable
    cellular necrosis and scarring to all major organs of the
    body in which cells will not regenerate, including the
    brain, spinal cord, eyes, lung, heart, liver, pancreas,
    kidney, adrenal, and blood vessels. If the disease is
    diagnosed during the first Stage, hospitalization is
    usually unnecessary, but allergies and asthma should be
    monitored closely. If the disease is diagnosed during the
    second Stage and even at the transition from the second to
    third Stages, early hospitalization may preserve the
    patient�s life. If however, the disease is only detected
    during the third Stage, death cannot be prevented in most
    Cases.

    Leave a comment:


  • widow1
    replied
    Sailor's family finds how dangerous mold can be in...

    Nevada.

    ****VIKTORIA PEARSON, vpearson@lahontanvalleynews.com
    April 8, 2007

    How much can one mother take is a question Michelle Bodenhorn of Fallon continues to ask herself each day while she sits in her 2-year-old daughter Elizabeth's hospital room at Renown Regional Medical Center in Reno.

    Bodenhorn was told in February that she needed to leave her home and all the family's possessions behind because of a mold problem caused by a water leak. ****

    http://www.lahontanvalleynews.com/ar...News/104080040

    Leave a comment:


  • widow1
    replied
    Walter Reed Woes Greater than Bldg 18

    Didn't post this within other article for fear y'all would miss;
    http://prorev.com/2007/03/walter-ree...hout-whole.htm

    Be sure to view the video link at bottom of article.
    Last edited by widow1; 03-21-2007, 19:50. Reason: missing link (I am ;)

    Leave a comment:


  • widow1
    started a topic Mold Info

    Mold Info

    I believe that one of the reasons some of the illnesses that the Viet Vets, in country, have been so difficult to get a handle on is the synergistic impact of the exposure to the rain agents and exotic molds. The fungii, already opportunistic pathogens, were given a big "come on in" to the body stripped of
    its normal immune defenses due to exposure to the chemical agents.

    This site has links to simply listed, well-known symptoms of mold exposure;
    http://mycotoxicosis.com/.

    Fungii are little understood due to their immense numbers of species and variations within those species. At any given time or stage of their life cycle, these pathogens produce different toxins as do each specific specie produce numerous toxins. Very complicated, but the effects are well known and documented.

    Denver, CO;

    VA Hospital turning away some patients due to aspergillus.
    http://www.9news.com/news/article.aspx?storyid=65931

    Check out the '04 report on the Denver facility. Remember, the hospital knew there were problems with aspergillosis in '01.

    http://www.va.gov/oig/publications/reports-list.asp


    Amazing that the Senate and House Veterans Affairs Committee members and their staffs missed those OIG reports.

    http://veterans.senate.gov/
    http://veterans.house.gov/index.htm

    More Veterans' Hospitals in the news;

    http://www.consortiumnews.com/2007/030807b.html

    http://www.dailymail.com/story/News/...vets+a+tragedy

    Here is one I missed from Amarillo regarding the VA Hospital there;
    http://www.kvii.com/news/news_story.aspx?id=28702

    And another about all VA hospitals and Kiley;
    http://www.mysanantonio.com/opinion/...d.297e5ef.html

    Must read; http://www.boston.com/news/education...est_practices/

    http://www.kaisernetwork.org/daily_r...fm?DR_ID=43509

    http://www.capitalnews9.com/content/...7050&SecID=285
    Last edited by widow1; 03-12-2007, 18:17. Reason: All related to same subject from same contributer.
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